Clinical Neuroscience

[Induced paranoid psychosis]

HAITS Géza1

NOVEMBER 01, 1965

Clinical Neuroscience - 1965;18(11)

[In addition to a brief review of the relevant literature, the author analyses two separate cases of induced paranoid psychosis in a family (mother-in-law, husband, wife, son) and a married couple. He points out the role of environmental influences and personality structure. He draws attention to the reflection of the superstitious-mystical social transmission (witchcraft) in the content of the delusions, despite the proximity of the capital, and the importance of the irrational element in the functioning of the suggestive mechanisms. ]

AFFILIATIONS

  1. Budapesti Orvostudományi Egyetem Psychiatriai Klinikája

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[Based on our analysis of one hundred patients with mittening, it seems that mittening sleep is indicative of a personality disorder manifested by recurrent psychic decompensations and conduct disorder at the level of organic cerebral lesions. Mitten sleep is not a disorder associated with a single clinical pathology unless it occurs in combination with the following factors : 1. a personality disorder with intermittent neurotic or psychotic decompensations or behavioural disturbances. 2. pathological vegetative phenomena. 3. diffuse cerebral lesions (ventricular dilatation and/or cortical atrophy). 4. minimum age : 15-34 years. ]

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[Authors have detected the same disease in a pair of 32 and 20-year-old brothers. Both eyes showed chronic uveitis, acute iritis, inveterate chorioretinitis nodules. One eye of the younger boy was already completely amaurotic. The latter also had meningoencephalitis, which was not favorably affected by cortisone. No virus was isolated from fresh mouth aphthae. Based on the literature and our own observations, the neurological symptoms were presumably allergic in origin.]

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[The high sensitivity of the central nervous system to decompression trauma is evident, and damage can occur not only during manifest neurological disease, but also latently, behind the onset of other forms of disease, or even without any manifest symptoms of decompression. The only effective causal therapy for decompression sickness of the central nervous system is recompression, carried out in a timely manner, with great care and, if necessary, with persistent patience. After unsuccessful recompression, the prospects for recovery are poor; permanent damage is caused and can lead to both somatic and functional psychiatric pathologies. In view of the risk of serious and permanent neurological lesions, the most careful prevention and therapy is essential. Among the preventive measures to be considered is limiting the length of time spent under hyperbaric pressure to prevent saturation of the slowly saturating lipid-containing tissues. The rate of evisceration should be strictly adhered to as prescribed. The implementation of recompression is the decisive factor in the therapy: after successful recompression, very slow decompression and, if necessary, oxygen inhalation should be used. In the compensation of occupational diseases in caisson workers, the possibility of an organic origin of functional conditions should also be considered. ]

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[Authors have observed spinal paralysis in a cat caused by 220 V electric shock. The condition is thought to be related to Panse's spinal atrophic paralysis. The clinical presentation of the case is supported by histopathological studies. The expected neurological consequences of electrocution are reviewed, and the pathology and pathomechanism of electrocution are discussed. The development of Panse's syndrome is associated with the specific vascularity of the spinal cord, based on the conclusions drawn from their case. We would like to thank Ernő Guzsal, assistant professor, for the preparation of the microphotographs. ]

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