Hungarian Radiology

[Initial experiencies with sonoelastography in the examinations of breast diseases]

BORBOLA György, KARDOS Klára, TASNÁDI Tünde

MARCH 22, 2008

Hungarian Radiology - 2008;82(01-02)

[INTRODUCTION - Recently the ultrasound examination of the strain of circumscribed breast diseases has been introduced in the non-invasive breast examination, called sonoelastography. In Hungary, the authors had the first possibility to start with this method. They report on their initial experiences. MATERIAL AND METHODS - 61 circumscribed breast lesions in 41 patients sonoelastographic examinations were performed by Hitachi EUB 6500 system using a EZUTE3 real-time elastography unit. 48 lesions strain-ratio was calculated. 22 masses were verified pathologically (18 benign and 4 malignant) and 39 were considered benign upon the findings of clinical mammography and by follow up. They classified them on the basis of patterns published by Itoh, et al. The examinations were done by three experienced radiologists. The classification was done by consensus. RESULT - The all lesions which were verified pathologically or on the basis of examinations or follow ups were thought benign showed the pattern type from 1 to 3. Most of the cystic lesions showed the streaky cystic pattern. There were small number of malignant lesions in their material, and all of them gave the elastic pattern of 4 and 5. The numbers of strain-ratio of zone lesions with pattern 3 overlapping with the lesions of malignant ones. CONCLUSION - The first results showed that both the coloured elastographic pattern and the quantitative strainratio could be used well in the non-invasive diagnostic procedure of breast lesions. It could increase the diagnostic safety. Larger number of examinations are necessitated to find the exact diagnostic role of this method.]

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Hungarian Radiology

[Use of covered stents in the endovascular treatment of extracranial stenosis of the internal carotid artery]

SZÓLICS Alex, VÖRÖS Erika, SZTRIHA László, SZIKRA Péter, SZÓLICS Miklós, PALKÓ András

[INTRODUCTION - Significant stenosis of the internal carotid artery is frequently treated with stent placement. With growing clinical experience and usage of finer instrumentation, the incidence of periprocedural complications have reduced in larger centers. Two-thirds of the complications are postprocedural, due to the embolisation through the stent structure. Covered stents seem to be a good option against such embolisation. Our study demonstrates the efficiency, safety and feasibility of covered stent grafts, and the long term outcome of patients who underwent endovascular treatment of extracranial internal carotid artery stenoses, caused by highly embologenic plaques. MATERIALS AND METHODS - Between 2002 and 2003, 30 patients (22 male, 8 female, aged 50-89yrs, mean: 66 yrs) with 30 internal carotid artery stenoses having ipsilateral symptoms and/or stenotic lesions caused by irregular or ulcerated soft plaques or restenosis were treated with self-expanding covered stents (Symbiot, Boston Scientific). Predilatation and protecting devices were not used. Postdilatation was applied in every patient. Mean followup was 60 months (range 57-66 months), by Doppler ultrasonography as well as clinical examination. RESULTS - The degree of stenosis was found to range from 70% to subtotal occlusion. The plaque surfaces were irregular or ulcerated in 70%. The stenotic lesions were up to 30 mm in length. The narrowing of the internal carotid artery never extended to the common carotid artery. The technical success rate of stenting was 100%. The stents could be positioned with an accuracy of 2-3 mm. Periprocedurally, there were no neurological complications or deaths. During follow-up no strokes or stroke-related deaths occurred. Restenosis was found in two patients (6,6%) who underwent successful balloon dilatation. CONCLUSION - Our experience indicates that the covered stent is an efficient periprocedural and postprocedural “protecting device” to prevent neurological complications due to embolizations caused by high-risk plaques in stenotic lesions of extracranial internal carotid artery.]

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LOMBAY Béla

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KISS-TÓTH Emőke, KUN Gáborné, LOMBAY Béla

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