Clinical Neuroscience

[Extra., intracranialis localised cavernosus haemangioma]

PÁSZTOR Emil1, SZABÓ Gyula1, SLOWIK Felicia1, ZOLTÁN János1

OCTOBER 01, 1963

Clinical Neuroscience - 1963;16(10)

[ In a 36-year-old female patient, multiple cavernous haemangiomas were detected and histologically verified in the right cheek, right os parietalera and right skull base. The case cannot be classified as a multiple haemangiomatous syndrome (Willis), the lesions are localized to the right external carotid artery system. Two years previously, plastic surgery of the right facial skin was performed and currently a large non-bony tumour extending from the base of the skull into the intracranial space has been removed. We discuss the pathological, angiographic and surgical indicative aspects of cavernous haemangiomas of the skull bone in our case. ]


  1. Országos Idegsebészeti Tudományos Intézet és Központi Honvéd Kórház



Further articles in this publication

Clinical Neuroscience

[Extracranial electrostimulation I. Effect of electrostimulation on the eeg]

HASZNOS Tivadar, FENYŐ Egon, ANTAL János

[The authors used electrodes placed on the scalp to stimulate the scalp partly locally and partly bitemporally with rectangular current pulses. Local stimulation was also performed with bipolar electrode placement. The frequency of stimulation was varied between 2/sec and 10 000/sec and the duration between 0.03 msec and 100 msec. The applied voltage was usually 30 V. The stimulation lasted for 30 or 60 seconds. EEG recording was performed immediately after the end of stimulation. The tests were performed on 18 patients. In patients with tumours, an increase in slow activity and focal excitatory signals were observed in response to local stimulation. In temporal epilepsy an increase in focal symptoms and in post-traumatic patients an increase in slow focal activation were observed. In patients with centrencephalic epilepsy with bitemporal stimulation, centrencephalic paroxysms were seen, and in temporal epilepsy, local slow and excitatory activation of signs. The authors hypothesize that local stimulation is primarily used to stimulate cortical and cortical proximal structures, whereas bitemporal electrostimulation seems to be a suitable technique for stimulation of the mesodiencephalon. ]

Clinical Neuroscience

[Relationships between EEG and motor responses in Evipan anaesthesia]


[In humans, the more profound the anaesthesia, the more the "pure synchronisation" of the EEG responses to different peripheral stimuli becomes apparent. Biological signs of reduced sleep depth and pupil dilation are associated with a more sustained synchronisatio, which therefore corresponds to an "inverse" electrical manifestation of a coarse, limited arousal. However, as anaesthesia wears off, there is an increasing proportion of more rapid frequencies in the EEG responses. At first, desynchronised sleep stages appear in which the spontaneous EEG, bioelectrical and motor responses resemble the 1st (IV) stage of human spontaneous sleep and the "paradoxical" ("rhombencephalic") sleep stages of animals. The development of such "paradoxical" stages in humans is often facilitated by peripheral stimulus influx. Later on, bipartite (desynchronisatio + post-synchronisatio) responses are formed; the biological meaning and origin of "pure synchronisatio" and "post-synchronisatio" are not identical. The return of alert wakefulness occurs in the company of "pure desynchronisatio" responses. The generation of these response types is determined by states of functional disinhibition and restitution induced by anaesthesia, which reflect the relations of superiority and inferiority of dependence between reticular systems. The pupillomotor reactivity during barbiturate action is poor compared to the morphological sensitivity of the EEG; the synchronization induced by peripheral stimuli is accompanied by mydriasis, whereas the "paradoxical" phase is accompanied by miosis. The EDG is hyperactive during moderate barbiturate exposure but is rapidly extinguished by deepening anaesthesia; its return and increased excitability are linked to orientational responsiveness, minimal verbal contact ability and a phase of dual EEG responses.]

Clinical Neuroscience

[Constitution and Rules of the World Federation of Neurology (Fédération Mondiale de Neurologie). 1963. ]

[The author reports on the Constitution and Rules of the World Federation of Neurology (Fédération Mondiale de Neurologie). ]

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[The significance of hypertension as one of the major cardiovascular risk factor is unquestionable. By achieving target blood pressure values differentiated by age and comorbidities, the risk of cardiovascular events can be significantly reduced. However, it is essential to the quality of life the patient spends the extra years of life thus gained. This is a really complex issue affecting many co-disciplines, but one of the most important of these is the mental health, maintaining cognitive functions, and avoiding dementia. High blood pressure impairs the blood supply to the target organs, including the brain, by damaging the smooth muscle of the arteries and accelerating atherosclerosis, which increases the risk, the frequency and the severity of mental decline in proportion to the degree of tension. This means serious implications not only for the individual, but for the family and the society, as well. A particular contradiction is that treating blood pressure to the target range does not automatically means preserving cognitive functions and avoiding the risk of dementia. Meta-analyzes of large studies have shown differences between the individual antihypertensive groups have been confirmed in this respect as well. Inhibitors of the renin-angiotensin system and calcium antagonists – mainly dihydropyridines – appear to be a priority in this regard. The authors provide an overview of the relationship between hypertension and mental abilities, with a review of the literature on the effects of antihypertensive therapy, with particular reference to the effects on cognitive function and dementia. ]

Clinical Neuroscience

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Clinical Neuroscience

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