Clinical Neuroscience - 1966;19(09)

Clinical Neuroscience

SEPTEMBER 01, 1966

[Data on the psychiatric use of levomepromazine (Tisercin)]

BÖSZÖRMÉNYI Zoltán, SOLTI Gyöngyi

[Authors report on the psychiatric efficacy of LPZ in 900 patients on a course of LPZ treatment, the psychopathological changes it induces, the results of their combined treatments (LPZ+neuroleptic, LPZ+antidepressant) and the side effects observed. The very significant anxiolytic, adjuvant-antidepressant, analgesic and adaptation-promoting effects of LPZ in initial psychosedation are confirmed. Examples are given of the course of treatment. Some results of the relevant pharmacological, physiological and clinical literature are summarised. ]

HUN 1966;19(09)

Clinical Neuroscience

SEPTEMBER 01, 1966

[Evaluation of the operation of the Institutes for the Care of the Mentally Ill in 1965]

DUBOVITZ Dénes

[There is no doubt that the effective implementation of these tasks - especially in areas with relatively poorly provided medical hours - depends to a large extent on the further quantitative development of the network of Institutes for Neurological Care and the strengthening of the number of hours. In the context of the determined development of mental health care, steps have been and will continue to be taken to improve the framework of the Institutes for Neurological Diseases. Filling these frameworks with appropriate content is the task of the doctors of the Institutes of Mental Health and other collaborating institutions, and its fulfilment will be a major step forward on the road to the development of mental health care in the country. ]

HUN 1966;19(09)

Clinical Neuroscience

SEPTEMBER 01, 1966

[Comparative clinical and pathological observations on periodic ataxia]

KOMÁR József, KOMÁR Gyula

[Authors observed periodic ataxia in a male patient and a mother cat. The clinical observation period was 2 1/2 years for the male and 1/2 year for the cat. At autopsy of the mother cat, no abnormalities in the central nervous system were observed either macroscopically or microscopically; the cluster of symptoms was probably observed as a separate pathology. Comparative observations also lead to conclusions about the origin of the male patient's symptoms. As regards the pathomechanism, it is assumed that, in addition to "biochemical damage" to the cerebellum, a transient insufficiency of the vertebrobasilar arterial system is the cause of the clinical symptoms. Authors consider periodic ataxia as a distinct pathology based on their observations. ]

HUN 1966;19(09)

Clinical Neuroscience

SEPTEMBER 01, 1966

[Report on the joint scientific meeting of English and Hungarian neurologists]

W. R.

[The author reports on a joint scientific meeting of English and Hungarian neurologists held in London in June 1966.]

HUN 1966;19(09)