Clinical Neuroscience - 1953;6(04)

Clinical Neuroscience

NOVEMBER 20, 1953

[On the treatment and origin of acute life-threatening mental disorders (so-called acute fatal catatonia)]

NAGY Tibor

[1. Acute life-threatening insanity can be cured by early and cumulative electroshock treatment. 2. The syndrome can be understood as a cluster of symptoms of cortico-cerebrospinal-pituitary-adrenal dysfunction, which may be triggered by psychic, somatic and toxic effects. 3. Instead of the various names used in the literature - delirium acutum, amentia, catatonia perniciosa, azotaemic insanity, acute toxic psychosis, etc. - a more comprehensive name seems justified.]

HUN 1953;6(04)

Clinical Neuroscience

NOVEMBER 20, 1953

[The u. n. treatment of acute fatal catatonia electrospasm]

VARGHA Miklós, KOVÁCS Béla

[Attempts have been made to deconstruct the nosolgical entity of schizophrenia from several angles. A detailed differentiation on the basis of psychopathology may be useful in order to find a closer link between the diverse biochemical, pathophysiological, morphological sub-scores and the psychopathological picture. Morphological lesions (Miskolczy, Hechst (Horányi), Josephy, Fünfgeld), as Miskolczy emphasises, only provide a consistent picture in certain forms of schizophrenia. The relative intactness of the cerebral areas (Stief, Hechst (Horányi)) is in contrast to some of the clinical symptoms, so that we can say that morphological lesions, whether of the brain or liver (Gaupp), etc., have not been able to prove the unity of schizophrenia or to clarify the affiliation of the different forms. Biochemical and pathophysiological research is at the stage of data collection, as Riebelling, in his most recent summary paper, has pointed out.]

HUN 1953;6(04)

Clinical Neuroscience

NOVEMBER 20, 1953

[Data on the pathomechanism of ventricular diverticulus formation]

SOÓS Imre, MOHÁCSI Ildikó

[A case of a lateral ventricular diverticulum herniating into the cisterna ambiens and a case of multiple intracerebral ventricular diverticulosis were described. Cisterna ambiens diverticulum may form in adulthood on rigid hydrocephalus cerebrum, although it has also been described in children. It is essentially an infratentorial herniation of the retrosplenial gyrus in a slowly developing brain pressure gradient. A large differential between supratentorial and infratentorial pressures is a favourable condition for its development. It can be diagnosed in vivo only by ventriculography. It is to be distinguished from arachnoid cysts of the cisterna ambiens, which do not converge with the ventricular system and have no parenchyma or ependyma in their walls. Intracerebral diverticula may originate anywhere in the ventricular system of the juvenile hydrocephalus brain. The pathomechanism of their origin is due to the readiness of the severely oedematous parenchyma to infiltrate and secondary collapse into the ventricular system. (Weber and da Rugna: dissezierende intracerebrale Divertikel) The involvement of the dilated third ventricle in the cisternae is not a true diverticulum, but is notoriously common in hydrocephalus. The clinical significance of diverticulum formation is that it is a self-healing activity that eliminates obstruction to cerebrospinal fluid circulation and provides a route for the surgical resolution of occlusive hydrocephalus.]

HUN 1953;6(04)

Clinical Neuroscience

NOVEMBER 20, 1953

[Some aspects of "primary-infectious" polyradiculoneuritis ]

JUBA Adolf

[After presenting the clinical and histopathological findings of two quite acute (2 days and 7-8 days) fatal "ascending polyradiculoneuritis", the author deals with pathological issues, which, given the severity of the cases, the histopathology provides a good starting point. The focus of the tissue process falls on the area between the root nerve and the intervertebral duct. However, inflammatory infiltrates are initially very sparse, consist of lymphocytes throughout, and do not extend beyond the area of ​​parenchymal destruction; that is, they do not correspond to real but to "reparative inflammation." Morphologically verifiable parenchymal injury is also initially disproportionate to severe clinical relapses and is only approx. It unfolds in 1 week. All this is against the generally accepted origin of the virus; confirmation of the allergic origin raised by Pette may not be by histological but by clinical-laboratory tests.]

HUN 1953;6(04)

Clinical Neuroscience

NOVEMBER 20, 1953

[Foreign Language Summaries]

[A summary of the articles published in the issue in Russian and German]

HUN 1953;6(04)