Clinical Neuroscience - 1951;4(01)

Clinical Neuroscience

MARCH 05, 1951

[The role of the cerebral cortex in inducing a vegetative crisis during an electroshock-induced epileptic seizure]

ARTHUR Kreindler

[The Institute of Neurology of the Academy of the People's Republic of Romania "Ivan Petrovich Pavlov" has epilepsy as one of its priority topics. In their view, epileptic seizures have three manifestations, three different areas in which they manifest themselves: the sensory-motor area, the vegetative area and the humoral area. For the last two years we have been studying these three manifestations of epileptic seizures, or as we have called them: the three crises of the epileptic seizure course: the somatic, the vegetative and the humoral crises.]

HUN 1951;4(01)

Clinical Neuroscience

MARCH 05, 1951

[The issue of myelitis necroticans]

JUBA Adolf, PÉTERFAI János

[The authors have reviewed two recent cases of myelitis necroticans, addressing questions of pathogenesis and aetiology and whether it is a distinct disease. It is concluded that the clinical presentation is variable and that the newer cases rarely agree completely with the original description. Histopathologically, necrosis of certain sections of the spinal cord is present in all cases, but, unlike in Foix and Alajouanine, it does not extend to the central part of the spinal cord and may also start higher up outside the lumbar sections. In many cases, the extra- and intraspinal vascular flow originally described is absent or modified and less marked. The vascular flow cannot be considered to play a primary role in the pathogenesis, but is a phenomenon parallel to the destruction of the spinal cord parenchyma. In the pathology, the infectious-inflammatory nature, given the absence of true tissue inflammation even in acute cases, cannot be confirmed and the spinal cord involvement is probably the result of various toxic agents. Thus, from a pathological point of view, it is not possible to speak of a single disease and the name "necrotising myelopathy" is justified instead of "myelitis necroticans".]

HUN 1951;4(01)

Clinical Neuroscience

MARCH 05, 1951

[In the context of Echolalia aphasia]

SIMONYI Gusztáv

[In anatomically proven Pick's atrophy, spontaneous speech deterioration was accompanied by long-standing sensory aphasia and echolalia. The echolalia also included sentences in foreign languages that were incomprehensible to the patient. Remnants of categorical behaviour can be recognised in the patient's attitude towards his environment. It is significant in the genesis of echolalia that in such cases there is damage to both the frontal and temporal lobes. In pathological conditions, echolalia occurs when speech intensity is reduced on the one hand and word comprehension is impaired on the other. An analogy between forced echolalia and other frontal forced phenomena (e.g. forced grasping) seems likely.]

HUN 1951;4(01)

Clinical Neuroscience

MARCH 05, 1951

[Cured case of gustator hemihyperhidrosis with sympathectomy]

BEKÉNY György

[In a 29-year-old male patient, hyperhidrosis of the scalp and neck had persisted for 9 years in response to various stimuli, but most markedly to joint impulses. The hyperhidrosis was temporarily relieved by blockade with novocaine of the ggl. cervivale sup. and then surgically removed. This observation demonstrates that the efferent pathway of gustatory hyperhidrosis can be eliminated by sympathectomy, which is important evidence to refute the dual innervation hypothesis of sweat glands. ]

HUN 1951;4(01)